Fig. 2From: Prim-O-glucosylcimifugin enhances the antitumour effect of PD-1 inhibition by targeting myeloid-derived suppressor cellsDifferentially expressed genes of PMN-MDSCs in tumour-bearing mice are mainly enriched in proliferation and metabolism-related pathways. a Statistical analysis of upregulated biological processes of B16-F10 tumour-bearing PMN-MDSCs. b The upregulated genes associated with the cell cycle, cell division and metabolic process of B16-F10 tumour-bearing PMN-MDSCs. c Statistical analysis of the upregulated molecular function of B16-F10 tumour-bearing PMN-MDSCs. d The upregulated genes associated with oxidoreductase, NADH dehydrogenase and electron carrier activities. e KEGG analysis of the upregulated genes of B16-F10 tumour-bearing PMN-MDSCs (f). The protein–protein interaction networks of upregulated proteins of B16-F10 tumour-bearing PMN-MDSCs. Significantly changed proteins are correlated with cell metabolismBack to article page